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t(19;21)(q13.4;q22)

Clinics and Pathology

Disease acute non lymphocytic leukemia (ANLL) secondary to toxic exposure
Note only one case, but with features identical to 2 other cases: one case of t(1;21)(p36;q22), and one case of t(18;21)(q21;q22)
Phenotype / cell stem origin M2-ANLL
Etiology about 50 years after radiation exposure from nuclear explosion
Clinics pancytopenia preceeded leukemia
Evolution complete remission was obtained and the patient returned to the previous pancytopenia; subsequent relapse occurred

Genes involved and Proteins

Gene Name AMP19
Location 19q13.4
Gene Name AML1
Location 21q22
Dna / Rna transcription is from telomere to centromere
Protein contains a Runt domain and, in the C-term, a transactivation domain; forms heterodimers; widely expressed; nuclear localisation; transcription factor (activator) for various hematopoietic-specific genes

Result of the chromosomal anomaly

Hybrid gene
Description AMP-19 is fused to AML1 out of frame
  
Fusion Protein
Description truncated AML1 with the DNA binding domain, but not a transcriptional activation region
Oncogenesis could function as a dominant negative inhibitor of normal AML1
  

External links

Other databaset(19;21)(q13.4;q22) Mitelman database (CGAP - NCBI)
Other databaset(19;21)(q13.4;q22) CancerChromosomes (NCBI)

To be noted

Additional cases are needed to delineate the epidemiology of this rare entity:
you are welcome to submit a paper to our new Case Report section.

Bibliography

Cloning of an AML1 translocation in a novel syndrome of radiation-induced acute myeloid leukemia.
Hromas RA, Busse TM, Shopnick R, Jumean H, Bowers C, Richkind K
Blood. 1999 ; 94 (numero suppl 1).
 

Contributor(s)

Written02-2000Jean-Loup Huret

Citation

This paper should be referenced as such :
Huret JL . t(19;21)(q13.4;q22). Atlas Genet Cytogenet Oncol Haematol. February 2000 .
URL : http://AtlasGeneticsOncology.org/Genes/t1921ID1182.html

© Atlas of Genetics and Cytogenetics in Oncology and Haematology
indexed on : Wed Sep 24 21:07:39 2008


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