CEP85L::ROS1 (6q22)

Genes involved

CEP85L (6q22.31)

ROS1 (6q22.1)

Oncogenic mechanisms

Constitutive activation of ROS1 tyrosine kinase activity

Targeted therapy

Crizotinib

Entrectinib

Tumor type

Glioblastoma multiforme

angiosarcoma

Highly cited references

Pubmed IDYearTitleCitations
236376312013Breakpoint analysis of transcriptional and genomic profiles uncovers novel gene fusions spanning multiple human cancer types.98
348751332022Detection of gene mutations and gene-gene fusions in circulating cell-free DNA of glioblastoma patients: an avenue for clinically relevant diagnostic analysis.61
271533962016Analyzing Somatic Genome Rearrangements in Human Cancers by Using Whole-Exome Sequencing.57
301710482018Rare but Recurrent ROS1 Fusions Resulting From Chromosome 6q22 Microdeletions are Targetable Oncogenes in Glioma.51
289121532017Comprehensive Genomic Profiling of 282 Pediatric Low- and High-Grade Gliomas Reveals Genomic Drivers, Tumor Mutational Burden, and Hypermutation Signatures.33
369234352023Overcoming CEP85L-ROS1, MKRN1-BRAF and MET amplification as rare, acquired resistance mutations to Osimertinib.24

Bibliography

Pubmed IDPublication YearTitleAuthors
301710482018Rare but Recurrent ROS1 Fusions Resulting From Chromosome 6q22 Microdeletions are Targetable Oncogenes in Glioma.Davare MA, et al
236376312013Breakpoint analysis of transcriptional and genomic profiles uncovers novel gene fusions spanning multiple human cancer types.Giacomini CP, et al
242619842013Exploration of the gene fusion landscape of glioblastoma using transcriptome sequencing and copy number data.Shah N, et al
255005442015The landscape and therapeutic relevance of cancer-associated transcript fusions.Yoshihara K, et al
252044152014The landscape of kinase fusions in cancer.Stransky N, et al