t(5;7)(p13;p22) SDK1/AMACR

0000-00-00   Jean-Loup Huret  , Philippe Dessen  

(Note: the fusion was discovered by NGS. Many chromosome rearrangement mechanisms such as translocation, inversion,deletion or complex rearrangement may lead to the same fusion)

Note

Non-annotated chromosomal abnormality. Preliminary data :
if you are an author who wish to write a full paper/card on this translocation,go to u00a0How to contribute

Summary

Note

Non-annotated chromosomal abnormality. Preliminary data :
if you are an author who wish to write a full paper/card on this translocation,go to  How to contribute

Genes Involved and Proteins

Gene name

SDK1

Location

7p22.2

Gene name

AMACR

Location

5p13.2

Highly cited references

Pubmed IDYearTitleCitations
266289962015High frequency of the SDK1:AMACR fusion transcript in Chinese prostate cancer.6

Article Bibliography

Pubmed IDLast YearTitleAuthors
223494602012RNA-seq analysis of prostate cancer in the Chinese population identifies recurrent gene fusions, cancer-associated long noncoding RNAs and aberrant alternative splicings.Ren S et al

Citation

Jean-Loup Huret ; Philippe Dessen

t(5;7)(p13;p22) SDK1/AMACR

Atlas Genet Cytogenet Oncol Haematol. 0000-00-00

Online version: http://atlasgeneticsoncology.org/solid-tumor/106794/t(5;7)(p13;p22)-sdk1-amacr