SLC27A5 (solute carrier family 27 member 5)

2014-11-01  

Identity

HGNC
LOCATION
19q13.43
LOCUSID
ALIAS
ACSB,ACSVL6,BACS,BAL,FACVL3,FATP-5,FATP5,VLACSR,VLCS-H2,VLCSH2
FUSION GENES

Other Information

Locus ID:

NCBI: 10998
MIM: 603314
HGNC: 10999
Ensembl: ENSG00000083807

Variants:

dbSNP: 10998
ClinVar: 10998
TCGA: ENSG00000083807
COSMIC: SLC27A5

RNA/Proteins

Gene IDTranscript IDUniprot
ENSG00000083807ENST00000263093Q9Y2P5
ENSG00000083807ENST00000594786M0R075
ENSG00000083807ENST00000595851M0R075
ENSG00000083807ENST00000601355Q9Y2P5

Expression (GTEx)

0
50
100
150
200
250
300

Pathways

PathwaySourceExternal ID
Primary bile acid biosynthesisKEGGko00120
PPAR signaling pathwayKEGGko03320
Primary bile acid biosynthesisKEGGhsa00120
PPAR signaling pathwayKEGGhsa03320
Metabolic pathwaysKEGGhsa01100
Bile secretionKEGGko04976
Bile secretionKEGGhsa04976
Bile acid biosynthesis, cholesterol => cholate/chenodeoxycholateKEGGhsa_M00104
Bile acid biosynthesis, cholesterol => cholate/chenodeoxycholateKEGGM00104
Conjugated bile acid biosynthesis, cholate => taurocholate/glycocholateKEGGM00106
Conjugated bile acid biosynthesis, cholate => taurocholate/glycocholateKEGGhsa_M00106
MetabolismREACTOMER-HSA-1430728
Metabolism of lipids and lipoproteinsREACTOMER-HSA-556833
Bile acid and bile salt metabolismREACTOMER-HSA-194068
Synthesis of bile acids and bile saltsREACTOMER-HSA-192105
Synthesis of bile acids and bile salts via 7alpha-hydroxycholesterolREACTOMER-HSA-193368
Synthesis of bile acids and bile salts via 24-hydroxycholesterolREACTOMER-HSA-193775
Recycling of bile acids and saltsREACTOMER-HSA-159418
Insulin resistanceKEGGhsa04931

Protein levels (Protein atlas)

Not detected
Low
Medium
High

References

Pubmed IDYearTitleCitations
379575402024Loss of SLC27A5 Activates Hepatic Stellate Cells and Promotes Liver Fibrosis via Unconjugated Cholic Acid.3
380598272024Metabolic Enzyme SLC27A5 Regulates PIP4K2A pre-mRNA Splicing as a Noncanonical Mechanism to Suppress Hepatocellular Carcinoma Metastasis.1
379575402024Loss of SLC27A5 Activates Hepatic Stellate Cells and Promotes Liver Fibrosis via Unconjugated Cholic Acid.3
380598272024Metabolic Enzyme SLC27A5 Regulates PIP4K2A pre-mRNA Splicing as a Noncanonical Mechanism to Suppress Hepatocellular Carcinoma Metastasis.1
366352562023SLC27A5 promotes sorafenib-induced ferroptosis in hepatocellular carcinoma by downregulating glutathione reductase.19
381572552023Copper metabolism-related risk score identifies hepatocellular carcinoma subtypes and SLC27A5 as a potential regulator of cuproptosis.1
366352562023SLC27A5 promotes sorafenib-induced ferroptosis in hepatocellular carcinoma by downregulating glutathione reductase.19
381572552023Copper metabolism-related risk score identifies hepatocellular carcinoma subtypes and SLC27A5 as a potential regulator of cuproptosis.1
313670132020SLC27A5 deficiency activates NRF2/TXNRD1 pathway by increased lipid peroxidation in HCC.54
316025262020Hepatic FATP5 expression is associated with histological progression and loss of hepatic fat in NAFLD patients.26
313670132020SLC27A5 deficiency activates NRF2/TXNRD1 pathway by increased lipid peroxidation in HCC.54
316025262020Hepatic FATP5 expression is associated with histological progression and loss of hepatic fat in NAFLD patients.26
234158022013Genetic defects in bile acid conjugation cause fat-soluble vitamin deficiency.30
234158022013Genetic defects in bile acid conjugation cause fat-soluble vitamin deficiency.30
220899232012Bile acid-CoA ligase deficiency--a new inborn error of bile acid metabolism.17

Citation

Dessen P

SLC27A5 (solute carrier family 27 member 5)

Atlas Genet Cytogenet Oncol Haematol. 2014-11-01

Online version: http://atlasgeneticsoncology.org/gene/73380/slc27a5-(solute-carrier-family-27-member-5)