The genes involved in the translocation were, indeed, described in the most recent report.

BCL6 (B-Cell Lymphoma 6)

3q27.3

706 amino acids; composed of a NH2-term BTB/POZ domain (amino acids 1-130 (32-99 according to Swiss-Prot) which mediates homodimerization and protein-protein interactions with other corepressors (including HDAC1 and NCOR2/SMRT to constitute a large repressing complex, another transcription repression domain (191-386), PEST sequences (300-417) with a KKYK motif (375-379), and six zinc finger at the C-term (518-541, 546-568, 574-596, 602-624, 630-652, 658-681), responsible for sequence specific DNA binding. Transcription repressor; recognizes the consensus sequence: TTCCT(A/C)GAA (Albagli-Curiel, 2003). Role in germinal centers of lymphoid follicles. BCL6 prevents ATM and TP53 to induce apoptosis in response to DNA rearrangements such as somatic hypermutation and class switch recombination. Therefore essential for normal B cell development.

GAS5 (growth arrest-specific 5 (non-protein coding))

1q25.1

GAS5 exons do not encode a polypeptide product (small nucleolar RNA (snoRNA) sequence) of the 5TOP gene family; role in the regulation of cell growth; provokes growth arrest; overexpression of GAS5 transcripts is reported to induce apoptosis.